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Effexor (Venlafaxine)
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Effexor

Generic Effexor is a qualitative medication which is taken in treatment of panic disorder, anxiety and depression. Generic Effexor effectiveness is in balancing the brain. It is a SSNRIs (selective serotonin and norepinephrine reuptake inhibitors).

Other names for this medication:

Similar Products:
Lexapro, Celexa, Paxil, Desyrel, Cymbalta

 

Also known as:  Venlafaxine.

Description

Generic Effexor is a perfect remedy in struggle against panic disorder, anxiety. Its target is to treat depression. Generic Effexor effectiveness is in balancing the brain. It is a SSNRIs (selective serotonin and norepinephrine reuptake inhibitors).

Generic name of Generic Effexor is Venlafaxine.

Effexor is also known as Venlafaxine, Ventab, Efexor, Venlor, Venla, Venlift.

Brand names of Generic Effexor are Effexor, Effexor XR.

Dosage

Generic Effexor is available in tablets and capsules. Generic Effexor is taken orally with food.

Do not crush or chew it.

Take Generic Effexor at the same time every day with water.

If you want to achieve most effective results do not stop taking Generic Effexor suddenly.

Overdose

If you overdose Generic Effexor and you don't feel good you should visit your doctor or health care provider immediately.

Storage

Store at a controlled temperature between 20 and 25 degrees C (68 degrees and 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Effexor are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Do not take Generic Effexor if you are allergic to Generic Effexor components.

Do not take Generic Effexor if you are pregnant, planning to become pregnant, or are breast-feeding.

Do not take it if you are under 18.

Be careful with Generic Effexor if you take ketoconazole (such as Nizoral); other antidepressants (fluoxetine (such as Prozac), sertraline (such as Zoloft), amoxapine (such as Ascendin), paroxetine (such as Paxil), citalopram (such as Celexa), protriptyline (such as Vivactil), clomipramine (such as Anafranil), trimipramine (such as Surmontil), desipramine (such as Norpramin), escitalopram (such as Lexapro), fluvoxamine (such as Luvox), imipramine (such as Tofranil), amitriptyline (such as Elavil), nortriptyline (such as Pamelor)); imetidine ( such as Tagamet HB, Tagamet); tryptophan; zolmitriptan (such as Zomig); rizatriptan (such as Maxalt), almotriptan (such as Axert), frovatriptan (such as Frova), naratriptan (such as Amerge), sumatriptan (such as Imitrex); warfarin (such as Coumadin); risperidone (such as Risperdal) or haloperidol (such as Haldol), monoamine oxidase inhibitors (MAOIs).

Avoid alcohol.

Be careful when you are driving or operating machinery.

It can be dangerous to stop Generic Effexor taking suddenly.

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Antidepressant agents have been associated with gynecomastia, but evidence for a causal link is insufficient.

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Venlafaxine (VEN), a serotonin and noradrenaline reuptake inhibitor is being used as a drug of choice for treating clinical depression even during pregnancy. It is an important therapeutic option in the treatment of perinatal depression, but the effects of VEN on fetus and the newborn are uncertain. Therefore, present study was undertaken to investigate the safety of in-utero exposure to VEN in terms of developmental neurotoxicity and neurodegenerative potential by using prenatal rat model. The selected doses of VEN (25, 40 and 50mg/kg) were administered to pregnant rats from GD 5 to 19 through oral gavage. The fetal brains were dissected and processed for histopathological measurements of neocortical thickness that showed significant reduction. Considering vulnerability of immature brain to free radical injury, VEN exposed neocortices were tested for reactive oxygen species (ROS) levels which were significantly increased. As ROS play important role in the initiation of apoptotic mechanisms, we explored for in situ detection of apoptosis by confocal microscopy that showed enhanced apoptosis including chromatin condensation which was further reconfirmed by electron microscopy. Substantially increased levels of pro-apoptotic protein Bax and decreased levels of anti-apoptotic protein Bcl2 as shown by western blotting also supported the increased neuro-apoptotic degeneration. For further correlation of these findings, prenatally VEN exposed young-adult rat offspring were assessed for open field exploratory behavior that showed increased anxiety-like and stereotypic responses indicating disturbed neurobehavioral pattern. The study concludes that prenatal VEN exposure may primarily enhance ROS generation that plays a key role in regulating release of proapoptotic factors from mitochondria and thereby enhancing apoptotic neurodegeneration that affect proliferation, migration and differentiation of cells, resulting in neuronal deficits manifested as long term neurobehavioral impairments.

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Venlafaxine hydrochloride, a structurally novel antidepressant, is also the first nontricyclic serotonin/norepinephrine reuptake inhibitor. Although venlafaxine has an overall side effect and safety profile that is comparable to other newer antidepressants, it can cause both transient and sustained elevations of supine diastolic blood pressure (SDBP), probably the result of noradrenergic potentiation.

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To evaluate the efficacy, safety, and tolerability of flexible-dose venlafaxine ER compared with placebo in the short-term treatment of generalized social anxiety disorder and, secondarily, to compare paroxetine with venlafaxine ER and paroxetine with placebo.

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Case report and literature review.

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This study was performed on rats after chronic constriction injury (CCI) to the sciatic nerve. The von Frey and Hargreaves' tests were used to assess mechanical allodynia and thermal hyperalgesia, respectively, after intraplantar (ipl) or subcutaneous (sc) administration of amitriptyline, doxepin, or venlafaxine, or their ipl co-administration with morphine on day 12-16 after injury.

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Amitriptyline up-regulated SOD1 messenger RNA in a time- and dose-dependent manner. The greatest up-regulation was following incubation with 10 mumol/L amitriptyline for 48 hours. The addition of bupropion, doxepin or venlafaxine to PC12 cell cultures also up-regulated SOD1 mRNA.

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Nearly 70% of participants had anxious depression. Remission rates were significantly lower and ratings of adverse event frequency were significantly greater in patients with anxious TRD than in those with nonanxious TRD. Presence of anxious depression predicted worse outcomes.

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Heart rate variability (HRV) reflects the cardiac autonomic regulation, and reduced HRV is considered a pathophysiological link between depression and cardiovascular mortality. So far, there is only limited information on the effects of venlafaxine and mirtazapine on HRV.We studied 28 nondepressed controls and 41 moderately depressed patients being treated with venlafaxine (n = 20) and mirtazapine (n = 21). Heart rate, blood pressure, and HRV were measured after a 6-day washout as well as after 14 and 28 days of treatment in supine and upright position.We found increased heart rate and reduced HRV in the depressed patients compared with the nondepressed controls. Moreover, HRV total power declined during the treatment period. Medication and remission status after 4 weeks were not related to the change in HRV.We conclude that depression is related to reduced HRV, which might reflect sympathovagal dysbalance. The widely used antidepressants venlafaxine and mirtazapine led to further decline in HRV. Clinicians should consider HRV effects in the selection of antidepressants.

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effexor 175 mg 2017-10-19

The patient achieved buy effexor pain relief through treatment with calcium, vitamin D, calcitonin, weekly alendronate, and venlafaxine for depression.

effexor user reviews 2015-07-20

The cytochrome P450 2D6 (CYP2D6) enzyme is responsible for metabolizing approximately 25% of pharmaceutical agents. Individuals with impaired CYP2D6 metabolism and those concomitantly receiving agents that inhibit CYP2D6 can have variations in concentrations of such medications and their metabolites buy effexor .

effexor 75mg reviews 2015-08-28

In this sample of 17 patients, the mean age was 65.6 years and 77% were female. Most strikingly, 53% of patients presented with late-onset atypical depression defined as first episode after the age of 50. Fifteen of the 17 patients (88%) completed the eight-week treatment trial. The mean score on the HRSD 24-item decreased from 22.2 +/- 5.1 at baseline to 11.8 +/- 8.9 (p<0.001), and the mean total atypical item score decreased from 6.2 +/- 1.6 to 2.8 +/- 2.0 (p < 0.001). Remission was defined as a final HRSD < or = 10 and a 50% reduction in baseline HRSD score. The intent-to Evista Medication Dosage -treat remission rate was 65% and the completer remission rate was 73%.

effexor 150 mg 2017-12-17

Serotonin and norepinephrine reuptake inhibitors (SNRIs) are antidepressants which have high affinity to both serotonin transporter (SERT) and norepinephrine transporter (NET). In studies in vitro, SNRIs have been reported to show a large variability in the affinity ratio between SERT Protonix Neonatal Dosing and NET. For instance, the reported affinity ratio is about 30 for venlafaxine and 1.6 for milnacipran. In this study in nonhuman primates, we aimed to investigate the relationship between SERT and NET affinity by measuring the in vivo occupancy at both transporters of venlafaxine and milnacipran.

effexor drug interactions 2016-09-07

We conducted a randomized, double-blind, placebo-controlled study of the efficacy and safety of Suprax Suspension Coupon once-daily venlafaxine extended release (XR) and fluoxetine in outpatients with major depression and concomitant anxiety.

effexor overdose death 2016-07-26

Although there were no differential treatment effects on parent or adolescent-report of conflict, remission was associated with improvement in the CBQ-P. In general, intake family conflict Celebrex 60 Mg did not predict remission, except in the sub-group of participants whose parents reported clinically significant parent-child conflict at intake, for whom high levels of parent-reported conflict predicted a lower likelihood of remission. Conflict also did not moderate treatment response.